Associated Papers
HYTANE-Identified Latrophilin-3 Cleavage by Meprin β Leads to Loss of the Interaction Domains.
Armbrust F et al.
Journal of Proteome Research, 2025
GPR37 is processed in the N-terminal ectodomain by ADAM10 and furin.
Mattila SO et al.
Faseb Journal : Official Publication of the Federation of American Societies for Experimental Biology, 2021
ADAM10 missense mutations potentiate β-amyloid accumulation by impairing prodomain chaperone function.
Suh J et al.
Neuron, 2013
ADAM10 prodomain mutations cause late-onset Alzheimer's disease: not just the latest FAD.
Vassar R
Neuron, 2013
Potential late-onset Alzheimer's disease-associated mutations in the ADAM10 gene attenuate {alpha}-secretase activity.
Kim M et al.
Human Molecular Genetics, 2009
A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model.
Postina R et al.
The Journal of Clinical Investigation, 2004
Alpha-secretase activity of the disintegrin metalloprotease ADAM 10. Influences of domain structure.
Fahrenholz F et al.
Annals of the New York Academy of Sciences, 2000
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