Associated Papers
Truncated variant rs373056577 confers increased risk of type 2 diabetes and missense variant rs121912717 is associated with hypertriglyceridemia in Bangladeshi population.
Sayed S et al.
Metabolism Open, 2025
In the context of the triple burden of malnutrition: A systematic review of gene-diet interactions and nutritional status.
Tan PY et al.
Critical Reviews in Food Science and Nutrition, 2024
Dietary acid load modifies the effects of ApoA2-265 T > C polymorphism on lipid profile and serum leptin and ghrelin levels among type 2 diabetic patients.
Abaj F et al.
Bmc Endocrine Disorders, 2022
Interaction between Apo A-II -265T > C polymorphism and dietary total antioxidant capacity on some oxidative stress and inflammatory markers in patients with type 2 diabetes mellitus.
Jafari Azad B et al.
The British Journal of Nutrition, 2022
ApoA2-256T > C polymorphism interacts with Healthy Eating Index, Dietary Quality Index-International and Dietary Phytochemical Index to affect biochemical markers among type 2 diabetic patients.
Esmaeily Z et al.
The British Journal of Nutrition, 2022
Deletion allele of Apo B gene is associated with higher inflammation, oxidative stress and dyslipidemia in obese type 2 diabetic patients: an analytical cross-sectional study.
Mokhtary N et al.
Bmc Endocrine Disorders, 2022
Genetic polymorphisms associated with obesity in the Arab world: a systematic review.
Younes S et al.
International Journal of Obesity (2005), 2021
A personalised diet study: The interaction between ApoA2 -265T > C polymorphism and dietary inflammatory index on oxidative and inflammatory markers and lipid profile in patients with type 2 diabetes mellitus: A cross-sectional study.
Karimi E et al.
International Journal of Clinical Practice, 2021
The interaction between ApoA2 -265T>C polymorphism and dietary fatty acids intake on oxidative stress in patients with type 2 diabetes mellitus.
Zamani E et al.
European Journal of Nutrition, 2017
The effect of weight loss on HDL subfractions and LCAT activity in two genotypes of APOA-II -265T>C polymorphism.
Moradi M et al.
Nutrition Journal, 2017
Apolipoprotein A2 -265 T>C polymorphism interacts with dietary fatty acids intake to modulate inflammation in type 2 diabetes mellitus patients.
Keramat L et al.
Nutrition (burbank, Los Angeles County, Calif.), 2017
APOA II genotypes frequency and their interaction with saturated fatty acids consumption on lipid profile of patients with type 2 diabetes.
Noorshahi N et al.
Clinical Nutrition (edinburgh, Scotland), 2016
APO A2 -265T/C Polymorphism Is Associated with Increased Inflammatory Responses in Patients with Type 2 Diabetes Mellitus.
Koohdani F et al.
Diabetes & Metabolism Journal, 2016
Interaction of dietary fat intake with APOA2, APOA5 and LEPR polymorphisms and its relationship with obesity and dyslipidemia in young subjects.
Domínguez-Reyes T et al.
Lipids in Health and Disease, 2015
APOA2 -256T>C polymorphism interacts with saturated fatty acids intake to affect anthropometric and hormonal variables in type 2 diabetic patients.
Basiri MG et al.
Genes & Nutrition, 2015
Evaluating the association of APOA2 polymorphism with insulin resistance in adolescents.
Zaki ME et al.
Meta Gene, 2014
Novel polymorphisms of the APOA2 gene and its promoter region affect body traits in cattle.
Zhou Y et al.
Gene, 2013
APOA2 Polymorphism in Relation to Obesity and Lipid Metabolism.
Zaki ME et al.
Cholesterol, 2013
Gene-Lifestyle Interactions in Obesity.
van Vliet-Ostaptchouk JV et al.
Current Nutrition Reports, 2012
Association between the APOA2 promoter polymorphism and body weight in Mediterranean and Asian populations: replication of a gene-saturated fat interaction.
Corella D et al.
International Journal of Obesity (2005), 2011
Adaptive genetic variation and heart disease risk.
Parnell LD et al.
Current Opinion in Lipidology, 2010
Effects of variations in the APOA1/C3/A4/A5 gene cluster on different parameters of postprandial lipid metabolism in healthy young men.
Delgado-Lista J et al.
Journal of Lipid Research, 2010
APOA2, dietary fat, and body mass index: replication of a gene-diet interaction in 3 independent populations.
Corella D et al.
Archives of Internal Medicine, 2009
An apolipoprotein A-II polymorphism (-265T/C, rs5082) regulates postprandial response to a saturated fat overload in healthy men.
Delgado-Lista J et al.
The Journal of Nutrition, 2007
The -256T>C polymorphism in the apolipoprotein A-II gene promoter is associated with body mass index and food intake in the genetics of lipid lowering drugs and diet network study.
Corella D et al.
Clinical Chemistry, 2007
Apolipoprotein A-II polymorphism and visceral adiposity in African-American and white women.
Lara-Castro C et al.
Obesity Research, 2005
Interaction between the LDL-receptor gene bearing a novel mutation and a variant in the apolipoprotein A-II promoter: molecular study in a 1135-member familial hypercholesterolemia kindred.
Takada D et al.
Journal of Human Genetics, 2002
Human evidence that the apolipoprotein a-II gene is implicated in visceral fat accumulation and metabolism of triglyceride-rich lipoproteins.
van 't Hooft FM et al.
Circulation, 2001