Associated Papers
Targeting natural splicing plasticity of APOBEC3B restricts its expression and mutagenic activity.
Rouf Banday A et al.
Communications Biology, 2021
33753867 (PubMed )
Supplement: 42003_2021_1844_MOESM3_ESM.xlsx › Supplementary_Data_2
Characterization of the mechanism by which the RB/E2F pathway controls expression of the cancer genomic DNA deaminase APOBEC3B.
Roelofs PA et al.
Elife, 2020
32985974 (PubMed )
Supplement: elife-61287-supp3.xls › S3_E2F_dereg_tumors
DNA mismatch repair promotes APOBEC3-mediated diffuse hypermutation in human cancers.
Mas-Ponte D et al.
Nature Genetics, 2020
32747826 (PubMed )
Supplement: EMS118039-supplement-Supplementary_Tables_1_10.xlsx › supplementaryTable5
Association of a germline copy number polymorphism of APOBEC3A and APOBEC3B with burden of putative APOBEC-dependent mutations in breast cancer.
Nik-Zainal S et al.
Nature Genetics, 2014
24728294 (PubMed )
Supplement: NIHMS57487-supplement-2.xlsx › SuppTable1B
APOBEC3B is an enzymatic source of mutation in breast cancer.
Burns MB et al.
Nature, 2013
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