Varaico: Literature-Extracted Genetic Variants

Gene:APOBEC3G
Variant:p.H186R
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APOBEC3G

NM_021822.4:p.H186R

Associated Papers

The APOBEC3G gene rs2294367(C>G) variant is associated with HIV-1 infection in Moroccan subjects.

Belbacha I et al.

Acta Tropica, 2023

Crystal Structure of a Soluble APOBEC3G Variant Suggests ssDNA to Bind in a Channel that Extends between the Two Domains.

Maiti A et al.

Journal of Molecular Biology, 2020

Different antiviral activities of natural APOBEC3C, APOBEC3G, and APOBEC3H variants against hepatitis B virus.

Kanagaraj A et al.

Biochemical and Biophysical Research Communications, 2019

Polymorphisms of the cytidine deaminase APOBEC3F have different HIV-1 restriction efficiencies.

Mohammadzadeh N et al.

Virology, 2019

Structural Determinants of the APOBEC3G N-Terminal Domain for HIV-1 RNA Association.

Fukuda H et al.

Frontiers in Cellular and Infection Microbiology, 2019

APOBEC3G expression and HIV-1 infection in Burkina Faso.

Compaore TR et al.

Journal of Public Health in Africa, 2018

Occurrence of APOBEC3G variations in West Indian HIV patients.

Singh H et al.

Microbial Pathogenesis, 2018

Correlation of Apolipoprotein B mRNA-editing Enzyme, Catalytic Polypeptide- like 3G Genetic Variant rs8177832 with HIV-1 Predisposition in Pakistani Population.

Iqbal K et al.

Current Hiv Research, 2018

DNA mutagenic activity and capacity for HIV-1 restriction of the cytidine deaminase APOBEC3G depend on whether DNA or RNA binds to tyrosine 315.

Polevoda B et al.

The Journal of Biological Chemistry, 2017

Association between polymorphisms of the APOBEC3G gene and chronic hepatitis B viral infection and hepatitis B virus-related hepatocellular carcinoma.

He XT et al.

World Journal of Gastroenterology, 2017

Association of single nucleotide polymorphisms of APOBEC3G with susceptibility to HIV-1 infection and disease progression among men engaging in homosexual activity in northern China.

Li Q et al.

Archives of Virology, 2017

Functional characterization of Vif proteins from HIV-1 infected patients with different APOBEC3G haplotypes.

Reddy K et al.

Aids (london, England), 2016

Coding region variant 186H/R in Exon 4 of APOBEC3G among individuals of Western India.

Singh H et al.

Apmis : Acta Pathologica, Microbiologica, Et Immunologica Scandinavica, 2016

Role of APOBEC3F Gene Variation in HIV-1 Disease Progression and Pneumocystis Pneumonia.

An P et al.

Plos Genetics, 2016

APOBEC3G Variants and Protection against HIV-1 Infection in Burkina Faso.

Compaore TR et al.

Plos One, 2016

Polymorphic APOBEC3 modulates chronic hepatitis B in Moroccan population.

Ezzikouri S et al.

Journal of Viral Hepatitis, 2013

Identification and antiviral activity of common polymorphisms in the APOBEC3 locus in human populations.

Duggal NK et al.

Virology, 2013

Genetic variants in the host restriction factor APOBEC3G are associated with HIV-1-related disease progression and central nervous system impairment in children.

Singh KK et al.

Journal of Acquired Immune Deficiency Syndromes (1999), 2013

Evidence for selection at HIV host susceptibility genes in a West Central African human population.

Zhao K et al.

Bmc Evolutionary Biology, 2012

HLA-DP gene polymorphisms and hepatitis B infection in the Japanese population.

Migita K et al.

Translational Research : the Journal of Laboratory and Clinical Medicine, 2012

Association of APOBEC3G genotypes and CD4 decline in Thai and Cambodian HIV-infected children with moderate immune deficiency.

Bunupuradah T et al.

Aids Research and Therapy, 2012

APOBEC3-mediated editing in HIV type 1 from pediatric patients and its association with APOBEC3G/CUL5 polymorphisms and Vif variability.

De Maio FA et al.

Aids Research and Human Retroviruses, 2012

Immunogenetics of HIV and HIV associated tuberculosis.

Raghavan S et al.

Tuberculosis (edinburgh, Scotland), 2012

A positively selected APOBEC3H haplotype is associated with natural resistance to HIV-1 infection.

Cagliani R et al.

Evolution; International Journal of Organic Evolution, 2011

Effect of HIV-1 Vif variability on progression to pediatric AIDS and its association with APOBEC3G and CUL5 polymorphisms.

De Maio FA et al.

Infection, Genetics and Evolution : Journal of Molecular Epidemiology and Evolutionary Genetics in Infectious Diseases, 2011

Intensity of deoxycytidine deamination of HIV-1 proviral DNA by the retroviral restriction factor APOBEC3G is mediated by the noncatalytic domain.

Feng Y et al.

The Journal of Biological Chemistry, 2011

Loci polymorphisms of the APOBEC3G gene in HIV type 1-infected Brazilians.

Bizinoto MC et al.

Aids Research and Human Retroviruses, 2011

APOBEC3G expression is dysregulated in primary HIV-1 infection and polymorphic variants influence CD4+ T-cell counts and plasma viral load.

Reddy K et al.

Aids (london, England), 2010

Common genetic variation and the control of HIV-1 in humans.

Fellay J et al.

Plos Genetics, 2009

Host genetic determinants of human immunodeficiency virus infection and disease progression in children.

Singh KK et al.

Pediatric Research, 2009

Absence of H186R polymorphism in exon 4 of the APOBEC3G gene among North Indian individuals.

Rathore A et al.

Genetic Testing, 2008

Detecting AIDS restriction genes: from candidate genes to genome-wide association discovery.

Hutcheson HB et al.

Vaccine, 2008

Evolution of pharmacogenomics.

Somogy A

Proceedings of the Western Pharmacology Society, 2008

Genetic association of the antiviral restriction factor TRIM5alpha with human immunodeficiency virus type 1 infection.

Speelmon EC et al.

Journal of Virology, 2006

Exhaustive genotyping of the CEM15 (APOBEC3G) gene and absence of association with AIDS progression in a French cohort.

Do H et al.

The Journal of Infectious Diseases, 2005

APOBEC3G genetic variants and their influence on the progression to AIDS.

An P et al.

Journal of Virology, 2004

Predicting nsSNPs that Disrupt Protein-Protein Interactions Using Docking.

Goodacre N et al.

Ieee/acm Transactions on Computational Biology and Bioinformatics, 0

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Regarding the variants extracted from papers and shown text snippets from research papers, the following holds: "Some rights reserved. This work permits non-commercial use, distribution and reproduction in any medium, provided the original author and source are credited."