Associated Papers
Protein misfolding, aggregation and other factors determining the amyloidogenic landscape of human leucocyte chemotaxin-2: A review.
Kumar A et al.
Archives of Biochemistry and Biophysics, 2025
Apolipoprotein C-III amyloidosis in white lions (Panthera leo).
Kobayashi N et al.
Veterinary Pathology, 2024
Renal amyloidosis: an update on diagnosis and pathogenesis.
Gupta N et al.
Protoplasma, 2020
A transgenic mouse model reproduces human hereditary systemic amyloidosis.
Chabert M et al.
Kidney International, 2019
Proteomic Analysis for the Diagnosis of Fibrinogen Aα-chain Amyloidosis.
Taylor GW et al.
Kidney International Reports, 2019
Disruption of protein function by pathogenic mutations: common and uncommon mechanisms 1.
Taipale M
Biochemistry and Cell Biology = Biochimie Et Biologie Cellulaire, 2019
Hereditary Renal Amyloidosis Associated With a Novel Apolipoprotein A-II Variant.
Prokaeva T et al.
Kidney International Reports, 2017
Protein Misfolding, Amyloid Formation, and Human Disease: A Summary of Progress Over the Last Decade.
Chiti F et al.
Annual Review of Biochemistry, 2017
28498720 (PubMed )
Supplement: bi86_chiti_supmat.pdf
Renal Amyloidosis Associated With 5 Novel Variants in the Fibrinogen A Alpha Chain Protein.
Rowczenio D et al.
Kidney International Reports, 2017
Clinical, biopsy, and mass spectrometry characteristics of renal apolipoprotein A-IV amyloidosis.
Dasari S et al.
Kidney International, 2016
D25V apolipoprotein C-III variant causes dominant hereditary systemic amyloidosis and confers cardiovascular protective lipoprotein profile.
Valleix S et al.
Nature Communications, 2016
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