Associated Papers
A Global Approach to the Long-Term Follow-up of 17 Families with Bilateral Macronodular Adrenal Disease.
Charchar HLS et al.
The Journal of Clinical Endocrinology and Metabolism, 2025
Advancements in the research of the structure, function, and disease-related roles of ARMC5.
Qu Y et al.
Frontiers of Medicine, 2025
The mutational landscape of ARMC5 in Primary Bilateral Macronodular Adrenal Hyperplasia: an update.
Bouys L et al.
Orphanet Journal of Rare Diseases, 2025
ARMC5 mutations are associated with high levels of proliferating cell nuclear antigen and the presence of the serotonin receptor 5HT4R in PMAH nodules.
Conceição BBD et al.
Archives of Endocrinology and Metabolism, 2020
Cullin 3 targets the tumor suppressor gene ARMC5 for ubiquitination and degradation.
Cavalcante IP et al.
Endocrine-related Cancer, 2020
A multicenter experience on the prevalence of ARMC5 mutations in patients with primary bilateral macronodular adrenal hyperplasia: from genetic characterization to clinical phenotype.
Albiger NM et al.
Endocrine, 2017
Genetics of primary bilateral macronodular adrenal hyperplasia: a model for early diagnosis of Cushing's syndrome?
Drougat L et al.
European Journal of Endocrinology, 2015
ARMC5 mutations are a frequent cause of primary macronodular adrenal Hyperplasia.
Alencar GA et al.
The Journal of Clinical Endocrinology and Metabolism, 2014
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