Associated Papers
Atg16l1 promotes lung transplant tolerance by regulating glycolysis in macrophages.
Cano M et al.
Biorxiv : the Preprint Server for Biology, 2025
Balancing immunity: disease risk mutation can be beneficial.
Lin X et al.
Trends in Immunology, 2025
Heterozygosity for Crohn's disease risk allele of ATG16L1 promotes unique protein interactions and protects against bacterial infection.
Yao X et al.
Immunity, 2025
The Inflammatory Bowel Disease-Associated Autophagy Gene Atg16L1T300A Acts as a Dominant Negative Variant in Mice.
Gao P et al.
Journal of Immunology (baltimore, Md. : 1950), 2017
Myeloid ATG16L1 Facilitates Host-Bacteria Interactions in Maintaining Intestinal Homeostasis.
Zhang H et al.
Journal of Immunology (baltimore, Md. : 1950), 2017
ATG16L1: A multifunctional susceptibility factor in Crohn disease.
Salem M et al.
Autophagy, 2015
Atg16L1 T300A variant decreases selective autophagy resulting in altered cytokine signaling and decreased antibacterial defense.
Lassen KG et al.
Proceedings of the National Academy of Sciences of the United States of America, 2014
A Crohn's disease variant in Atg16l1 enhances its degradation by caspase 3.
Murthy A et al.
Nature, 2014
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