Associated Papers
Bi-allelic loss-of function variants in ATP2A1 cause autosomal recessive Brody disease.
Cao C et al.
Qjm : Monthly Journal of the Association of Physicians, 2025
High-yield heterologous expression of wild type and mutant Ca(2+) ATPase: Characterization of Ca(2+) binding sites by charge transfer.
Liu Y et al.
Journal of Molecular Biology, 2009
Functional and structural roles of critical amino acids within the"N", "P", and "A" domains of the Ca2+ ATPase (SERCA) headpiece.
Ma H et al.
Biochemistry, 2005
Ca2+ occlusion and gating function of Glu309 in the ADP-fluoroaluminate analog of the Ca2+-ATPase phosphoenzyme intermediate.
Inesi G et al.
The Journal of Biological Chemistry, 2004
Substrate-induced conformational fit and headpiece closure in the Ca2+ATPase (SERCA).
Ma H et al.
The Journal of Biological Chemistry, 2003
Detailed characterization of the cooperative mechanism of Ca(2+) binding and catalytic activation in the Ca(2+) transport (SERCA) ATPase.
Zhang Z et al.
Biochemistry, 2000
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