Associated Papers
The genomic landscape of cholangiocarcinoma reveals the disruption of post-transcriptional modifiers.
Zhang Y et al.
Nature Communications, 2022
35650238 (PubMed )
Supplement: 41467_2022_30708_MOESM1_ESM.pdf
ROBO1 p.E280* Loses the Inhibitory Effects on the Proliferation and Angiogenesis of Wild-Type ROBO1 in Cholangiocarcinoma by Interrupting SLIT2 Signal.
Zhou T et al.
Frontiers in Oncology, 2022
35615148 (PubMed )
Supplement: DataSheet_1.xlsx › Sheet1
Patient-derived models recapitulate heterogeneity of molecular signatures and drug response in pediatric high-grade glioma.
He C et al.
Nature Communications, 2021
34215733 (PubMed )
Supplement: 41467_2021_24168_MOESM5_ESM.xlsx › SD2d
Analysis of genetic alterations identifies the frequent mutation of GNAS in colorectal laterally spreading tumors.
Nong Y et al.
Cancer Communications (london, England), 2020
32762003 (PubMed )
Supplement: CAC2-40-636-s001.pdf
Extreme intratumour heterogeneity and driver evolution in mismatch repair deficient gastro-oesophageal cancer.
von Loga K et al.
Nature Communications, 2020
31949146 (PubMed )
Supplement: 41467_2019_13915_MOESM4_ESM.xlsx › Tumor_2
Type II enteropathy-associated T-cell lymphoma features a unique genomic profile with highly recurrent SETD2 alterations.
Roberti A et al.
Nature Communications, 2016
27600764 (PubMed )
Supplement: ncomms12602-s2.xlsx › Supplementary_Data_2
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