Associated Papers
The CaMKII D135N mutation blocks kinase activity and reduces GluN2B binding.
Larsen ME et al.
Biorxiv : the Preprint Server for Biology, 2025
NMDA Receptor-Arc Signaling Is Required for Memory Updating and Is Disrupted in Alzheimer's Disease.
Yang L et al.
Biological Psychiatry, 2023
Locally-Induced CaMKII Translocation Requires Nucleotide Binding.
Fitzgerald ZT et al.
Frontiers in Synaptic Neuroscience, 2020
The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targeting.
Tullis JE et al.
Plos One, 2020
Ca2+/Calmodulin-Dependent Protein Kinase II (CaMKII) β-Dependent Phosphorylation of GABAB1 Triggers Lysosomal Degradation of GABAB Receptors via Mind Bomb-2 (MIB2)-Mediated Lys-63-Linked Ubiquitination.
Zemoura K et al.
Molecular Neurobiology, 2019
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