Associated Papers
A CARMIL2 gain-of-function mutation suffices to trigger most CD28 costimulatory functions in vivo.
Zhang F et al.
The Journal of Experimental Medicine, 2025
Distinctive genomic features of human T-lymphotropic virus type 1-related adult T-cell leukemia-lymphoma in Western populations.
Myers CS et al.
Haematologica, 2024
TCR Pathway Mutations in Mature T Cell Lymphomas.
Zhang Y et al.
Journal of Immunology (baltimore, Md. : 1950), 2023
Deregulation of JAK2 signaling underlies primary cutaneous CD8+ aggressive epidermotropic cytotoxic T-cell lymphoma.
Bastidas Torres AN et al.
Haematologica, 2022
33792220 (PubMed )
Supplement: 2020_274506_TORRES_SUPPL.pdf
Integrated genomic analyses of cutaneous T-cell lymphomas reveal the molecular bases for disease heterogeneity.
Park J et al.
Blood, 2021
RLTPR Q575E: A novel recurrent gain-of-function mutation in patients with adult T-cell leukemia/lymphoma.
Uchida Y et al.
European Journal of Haematology, 2021
Contribution of STAT3 and RAD23B in Primary Sézary Cells to Histone Deacetylase Inhibitor FK228 Resistance.
Butler RM et al.
The Journal of Investigative Dermatology, 2019
Genomic analysis reveals recurrent deletion of JAK-STAT signaling inhibitors HNRNPK and SOCS1 in mycosis fungoides.
Bastidas Torres AN et al.
Genes, Chromosomes & Cancer, 2018
Genomic Analyses Identify Recurrent Alterations in Immune Evasion Genes in Diffuse Large B-Cell Lymphoma, Leg Type.
Zhou XA et al.
The Journal of Investigative Dermatology, 2018
Novel CARMIL2 Mutations in Patients with Variable Clinical Dermatitis, Infections, and Combined Immunodeficiency.
Alazami AM et al.
Frontiers in Immunology, 2018
Genomic analysis of 220 CTCLs identifies a novel recurrent gain-of-function alteration in RLTPR (p.Q575E).
Park J et al.
Blood, 2017
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