Associated Papers
Comparison of an In Vitro Diagnostic Next-Generation Sequencing Assay with Sanger Sequencing for HIV-1 Genotypic Resistance Testing.
Tzou PL et al.
Journal of Clinical Microbiology, 2018
Short Communication: HIV-1 Variants That Use Mouse CCR5 Reveal Critical Interactions of gp120's V3 Crown with CCR5 Extracellular Loop 1.
Platt EJ et al.
Aids Research and Human Retroviruses, 2015
Impact of the HIV integrase genetic context on the phenotypic expression and in vivo emergence of raltegravir resistance mutations.
Nguyen TT et al.
The Journal of Antimicrobial Chemotherapy, 2015
Elucidating a key anti-HIV-1 and cancer-associated axis: the structure of CCL5 (Rantes) in complex with CCR5.
Tamamis P et al.
Scientific Reports, 2014
Reversible and efficient activation of HIV-1 cell entry by a tyrosine-sulfated peptide dissects endocytic entry and inhibitor mechanisms.
Platt EJ et al.
Journal of Virology, 2014
Involvement of the second extracellular loop and transmembrane residues of CCR5 in inhibitor binding and HIV-1 fusion: insights into the mechanism of allosteric inhibition.
Maeda K et al.
Journal of Molecular Biology, 2008
Broad antiretroviral activity and resistance profile of the novel human immunodeficiency virus integrase inhibitor elvitegravir (JTK-303/GS-9137).
Shimura K et al.
Journal of Virology, 2008
An allosteric rheostat in HIV-1 gp120 reduces CCR5 stoichiometry required for membrane fusion and overcomes diverse entry limitations.
Platt EJ et al.
Journal of Molecular Biology, 2007
Changes in the V3 region of gp120 contribute to unusually broad coreceptor usage of an HIV-1 isolate from a CCR5 Delta32 heterozygote.
Gorry PR et al.
Virology, 2007
Structural and molecular interactions of CCR5 inhibitors with CCR5.
Maeda K et al.
The Journal of Biological Chemistry, 2006
Variants of human immunodeficiency virus type 1 that efficiently use CCR5 lacking the tyrosine-sulfated amino terminus have adaptive mutations in gp120, including loss of a functional N-glycan.
Platt EJ et al.
Journal of Virology, 2005
Adaptive mutations in the V3 loop of gp120 enhance fusogenicity of human immunodeficiency virus type 1 and enable use of a CCR5 coreceptor that lacks the amino-terminal sulfated region.
Platt EJ et al.
Journal of Virology, 2001
Frequent substitution polymorphisms in African green monkey CCR5 cluster at critical sites for infections by simian immunodeficiency virus SIVagm, implying ancient virus-host coevolution.
Kuhmann SE et al.
Journal of Virology, 2001
Cooperation of multiple CCR5 coreceptors is required for infections by human immunodeficiency virus type 1.
Kuhmann SE et al.
Journal of Virology, 2000
A critical site in the core of the CCR5 chemokine receptor required for binding and infectivity of human immunodeficiency virus type 1.
Siciliano SJ et al.
The Journal of Biological Chemistry, 1999
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