Associated Papers
Full-length haplotype reconstruction of CD36 by long-read sequencing: uncovers a novel structural variant.
Liang S et al.
Bmc Genomics, 2026
Haplotypes analysis reveals the genetic basis of type I CD36 deficiency.
Xia W et al.
Scientific Reports, 2024
[Molecular polymorphism Analysis on CD36 Deficiency among Platelet Blood Donors in Shenzhen].
Xu YP et al.
Zhongguo Shi Yan Xue Ye Xue Za Zhi, 2022
Platelet CD36 deficiency is present in 2.6% of Arabian individuals and can cause NAIT and platelet refractoriness.
Flesch BK et al.
Transfusion, 2021
[Construction of eukaryotic expression vector for human platelet CD36 gene 220C>T and 429+4insg variants and analysis of their expressions in HEK293T cells].
Xu X et al.
Zhonghua Yi Xue Yi Chuan Xue Za Zhi = Zhonghua Yixue Yichuanxue Zazhi = Chinese Journal of Medical Genetics, 2019
Evaluation of novel Parkinson's disease candidate genes in the Chinese population.
Chew EGY et al.
Neurobiology of Aging, 2019
Human knockouts and phenotypic analysis in a cohort with a high rate of consanguinity.
Saleheen D et al.
Nature, 2017
28406212 (PubMed )
Supplement: NIHMS857566-supplement-Suppl__Table_1.xlsx › Supplementary_Table_1
Discovery and functional prioritization of Parkinson's disease candidate genes from large-scale whole exome sequencing.
Jansen IE et al.
Genome Biology, 2017
Incidence and molecular basis of CD36 deficiency in Shanghai population.
Li R et al.
Transfusion, 2015
Integrated analysis of germline and somatic variants in ovarian cancer.
Kanchi KL et al.
Nature Communications, 2014
24448499 (PubMed )
Supplement: NIHMS551112-supplement-5.xlsx › S_Data_4
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