Associated Papers
Functional characterization of all CDKN2A missense variants and comparison to in silico models of pathogenicity.
Kimura H et al.
Elife, 2025
40238651 (PubMed )
Supplement: elife-95347-supp9.xlsx › Appendix_1-table_9
Functional characterization of all CDKN2A missense variants and comparison to in silico models of pathogenicity.
Kimura H et al.
Biorxiv : the Preprint Server for Biology, 2025
38234851 (PubMed )
Supplement: media-2.pdf
p16INK4A enhances the transcriptional and the apoptotic functions of p53 through DNA-dependent interaction.
Al-Khalaf HH et al.
Molecular Carcinogenesis, 2017
Regulatory mechanisms of tumor suppressor P16(INK4A) and their relevance to cancer.
Li J et al.
Biochemistry, 2011
GRIM-19 and p16(INK4a) synergistically regulate cell cycle progression and E2F1-responsive gene expression.
Sun P et al.
The Journal of Biological Chemistry, 2010
A common variant of CDKN2A (p16) predisposes to breast cancer.
Debniak T et al.
Journal of Medical Genetics, 2005
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