Associated Papers
KDM2A and KDM3B as Potential Targets for the Rescue of F508del-CFTR.
D'Amore C et al.
International Journal of Molecular Sciences, 2022
Novel characteristics of a misprocessed mutant HERG channel linked to hereditary long QT syndrome.
Ficker E et al.
American Journal of Physiology. Heart and Circulatory Physiology, 2000
Glycerol reverses the misfolding phenotype of the most common cystic fibrosis mutation.
Sato S et al.
The Journal of Biological Chemistry, 1996
CFTR: the nucleotide binding folds regulate the accessibility and stability of the activated state.
Wilkinson DJ et al.
The Journal of General Physiology, 1996
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