Associated Papers
The UPR-PERK pathway is not a promising therapeutic target for mutant SOD1-induced ALS.
Dzhashiashvili Y et al.
Neurobiology of Disease, 2019
Preserving neuromuscular synapses in ALS by stimulating MuSK with a therapeutic agonist antibody.
Cantor S et al.
Elife, 2018
Prion-like propagation of mutant SOD1 misfolding and motor neuron disease spread along neuroanatomical pathways.
Ayers JI et al.
Acta Neuropathologica, 2016
Nuclear localization of human SOD1 and mutant SOD1-specific disruption of survival motor neuron protein complex in transgenic amyotrophic lateral sclerosis mice.
Gertz B et al.
Journal of Neuropathology and Experimental Neurology, 2012
Neuron-specific expression of mutant superoxide dismutase is sufficient to induce amyotrophic lateral sclerosis in transgenic mice.
Jaarsma D et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 2008
Early signs of motoneuron vulnerability in a disease model system: Characterization of transverse slice cultures of spinal cord isolated from embryonic ALS mice.
Avossa D et al.
Neuroscience, 2006
Altered long-term corticostriatal synaptic plasticity in transgenic mice overexpressing human CU/ZN superoxide dismutase (GLY(93)-->ALA) mutation.
Geracitano R et al.
Neuroscience, 2003
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