Associated Papers
HSP90β prevents aging-related cataract formation through regulation of the charged multivesicular body protein (CHMP4B) and p53.
Fu JL et al.
Proceedings of the National Academy of Sciences of the United States of America, 2023
Charged multivesicular body protein 4b forms complexes with gap junction proteins during lens fiber cell differentiation.
Zhou Y et al.
Faseb Journal : Official Publication of the Federation of American Societies for Experimental Biology, 2023
Loss of fiber cell communication may contribute to the development of cataracts of many different etiologies.
Beyer EC et al.
Frontiers in Physiology, 2022
Adding Some "Splice" to Stress Eating: Autophagy, ESCRT and Alternative Splicing Orchestrate the Cellular Stress Response.
Habib E et al.
Genes, 2021
Broadening the Mutation Spectrum in GJA8 and CHMP4B: Novel Missense Variants and the Associated Phenotypes in Six Chinese Han Congenital Cataracts Families.
Wang X et al.
Frontiers in Medicine, 2021
Biology of Inherited Cataracts and Opportunities for Treatment.
Shiels A et al.
Annual Review of Vision Science, 2019
Mutation profiles of congenital cataract genes in 21 northern Chinese families.
Zhang XH et al.
Molecular Vision, 2018
Association of CHMP4B and autophagy with micronuclei: implications for cataract formation.
Sagona AP et al.
Biomed Research International, 2014
CHMP4B, a novel gene for autosomal dominant cataracts linked to chromosome 20q.
Shiels A et al.
American Journal of Human Genetics, 2007
Molecular characteristics of inherited congenital cataracts.
Huang B et al.
European Journal of Medical Genetics, 0
A charged multivesicular body protein (CHMP4B) is required for lens growth and differentiation.
Zhou Y et al.
Differentiation; Research in Biological Diversity, 0
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