Associated Papers
Is there a causal link between folate status and schizophrenia? Evidence from genetic association studies.
Yoo S et al.
Nutritional Neuroscience, 2025
Catechol-O-methyltransferase Val158Met genotype and the clinical responses to duloxetine treatment or plasma levels of 3-methoxy-4-hydroxyphenylglycol and homovanillic acid in Japanese patients with major depressive disorder.
Atake K et al.
Neuropsychiatric Disease and Treatment, 2015
Association of TMEM132D, COMT, and GABRA6 genotypes with cingulate, frontal cortex and hippocampal emotional processing in panic and major depressive disorder.
Inoue A et al.
International Journal of Psychiatry in Clinical Practice, 2015
COMT Val158Met, but not BDNF Val66Met, is associated with white matter abnormalities of the temporal lobe in patients with first-episode, treatment-naïve major depressive disorder: a diffusion tensor imaging study.
Hayashi K et al.
Neuropsychiatric Disease and Treatment, 2014
Development of a fingerprinting panel using medically relevant polymorphisms.
Cross DS et al.
Bmc Medical Genomics, 2009
19379518 (PubMed )
Supplement: 1755-8794-2-17-S2.doc
Polymorphisms in catechol-O-methyltransferase and methylenetetrahydrofolate reductase in relation to the risk of schizophrenia.
Muntjewerff JW et al.
European Neuropsychopharmacology : the Journal of the European College of Neuropsychopharmacology, 2008
Catechol-O-methyltransferase genotype is associated with plasma total homocysteine levels and may increase venous thrombosis risk.
Gellekink H et al.
Thrombosis and Haemostasis, 2007
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