Varaico: Literature-Extracted Genetic Variants

Gene:CR1
Variant:p.P301S
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CR1

NM_000651.6:p.P301S

Associated Papers

Protein kinase CK2α' as a dual modulator of immune signaling and synaptic dysfunction in Tauopathy.

White A et al.

Biorxiv : the Preprint Server for Biology, 2025

Protein kinase CK2α' as a dual modulator of neuroimmune signaling and synaptic dysfunction in Tauopathy.

White A et al.

Research Square, 2025

Human herpesvirus-associated transposable element activation in human aging brains with Alzheimer's disease.

Feng Y et al.

Alzheimer's & Dementia : the Journal of the Alzheimer's Association, 2025

Mitochondrial complex III-derived ROS amplify immunometabolic changes in astrocytes and promote dementia pathology.

Barnett D et al.

Biorxiv : the Preprint Server for Biology, 2024

Limitations of human tau-expressing mouse models and novel approaches of mouse modeling for tauopathy.

Sahara N et al.

Frontiers in Neuroscience, 2023

Crry silencing alleviates Alzheimer's disease injury by regulating neuroinflammatory cytokines and the complement system.

Zhu XC et al.

Neural Regeneration Research, 2022

Complement Activation in the Central Nervous System: A Biophysical Model for Immune Dysregulation in the Disease State.

Peoples N et al.

Frontiers in Molecular Neuroscience, 2021

Alzheimer's Retinopathy: Seeing Disease in the Eyes.

Mirzaei N et al.

Frontiers in Neuroscience, 2020

Targeting apolipoprotein E for treating Alzheimer's disease.

Bales KR et al.

Neuroscience Letters, 2019

Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy.

Wu T et al.

Cell Reports, 2019

Genetic Risk Factors for Alzheimer Disease: Emerging Roles of Microglia in Disease Pathomechanisms.

Takatori S et al.

Advances in Experimental Medicine and Biology, 0

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Regarding the variants extracted from papers and shown text snippets from research papers, the following holds: "Some rights reserved. This work permits non-commercial use, distribution and reproduction in any medium, provided the original author and source are credited."