Associated Papers
APC-related multiple salivary gland lesions: spatial transcriptomic analysis reveals progressive WNT activation.
Chan-Pak-Choon F et al.
The Journal of Pathology, 2025
40879644 (PubMed )
Supplement: PATH-267-241-s002.xlsx › Table_S6
WILMS TUMOR MUTATIONAL SUBCLASSES CONVERGE TO DRIVE CCND2 OVEREXPRESSION.
Xu L et al.
Medrxiv : the Preprint Server for Health Sciences, 2023
36778325 (PubMed )
Supplement: media-4.xlsx › Sheet1
Oncogenic Serine 45-Deleted β-Catenin Remains Susceptible to Wnt Stimulation and APC Regulation in Human Colonocytes.
Parker TW et al.
Cancers, 2020
32751567 (PubMed )
Supplement: cancers-12-02114-s001.zip › cancers-848197_Supplementary_Data_Files_Parker_Rudeen_Neufeld_11july20.xlsx › Ctnnb1_Mutations
Oncogenic Mutations in Armadillo Repeats 5 and 6 of β-Catenin Reduce Binding to APC, Increasing Signaling and Transcription of Target Genes.
Liu P et al.
Gastroenterology, 2020
The long tail of oncogenic drivers in prostate cancer.
Armenia J et al.
Nature Genetics, 2018
β-catenin contributes to lung tumor development induced by EGFR mutations.
Nakayama S et al.
Cancer Research, 2014
Recurrent somatic mutation in DROSHA induces microRNA profile changes in Wilms tumour.
Torrezan GT et al.
Nature Communications, 2014
SHP-1 inhibits β-catenin function by inducing its degradation and interfering with its association with TATA-binding protein.
Simoneau M et al.
Cellular Signalling, 2011
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