Associated Papers
Recruitment of mitochondria into apoptotic signaling correlates with the presence of inclusions formed by amyotrophic lateral sclerosis-associated SOD1 mutations.
Soo KY et al.
Journal of Neurochemistry, 2009
Different regulation of wild-type and mutant Cu,Zn superoxide dismutase localization in mammalian mitochondria.
Kawamata H et al.
Human Molecular Genetics, 2008
Neural mitochondrial Ca2+ capacity impairment precedes the onset of motor symptoms in G93A Cu/Zn-superoxide dismutase mutant mice.
Damiano M et al.
Journal of Neurochemistry, 2006
SOD1 activity and protective factors in familial ALS patients with L84F SOD1 mutation.
Curti D et al.
Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders : Official Publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases, 2002
Superoxide dismutase-1 mutation-related neurotoxicity in familial amyotrophic lateral sclerosis.
Shibata N et al.
Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders : Official Publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases, 2000
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