Associated Papers
Evidence of an unprecedented cytoplasmic function of DDX11, the Warsaw breakage syndrome DNA helicase, in regulating autophagy.
Bonavita R et al.
Autophagy, 2025
Targeting DDX11 promotes PARP inhibitor sensitivity in hepatocellular carcinoma by attenuating BRCA2-RAD51 mediated homologous recombination.
Cao K et al.
Oncogene, 2024
DDX11 loss causes replication stress and pharmacologically exploitable DNA repair defects.
Jegadesan NK et al.
Proceedings of the National Academy of Sciences of the United States of America, 2021
Warsaw Breakage Syndrome associated DDX11 helicase resolves G-quadruplex structures to support sister chromatid cohesion.
van Schie JJM et al.
Nature Communications, 2020
The iron-sulfur helicase DDX11 promotes the generation of single-stranded DNA for CHK1 activation.
Simon AK et al.
Life Science Alliance, 2020
Interaction of the Warsaw breakage syndrome DNA helicase DDX11 with the replication fork-protection factor Timeless promotes sister chromatid cohesion.
Cortone G et al.
Plos Genetics, 2018
A Long Noncoding RNA Regulates Sister Chromatid Cohesion.
Marchese FP et al.
Molecular Cell, 2016
Tim/Timeless, a member of the replication fork protection complex, operates with the Warsaw breakage syndrome DNA helicase DDX11 in the same fork recovery pathway.
Calì F et al.
Nucleic Acids Research, 2016
A distinct triplex DNA unwinding activity of ChlR1 helicase.
Guo M et al.
The Journal of Biological Chemistry, 2015
Biochemical characterization of Warsaw breakage syndrome helicase.
Wu Y et al.
The Journal of Biological Chemistry, 2012
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