Associated Papers
Lack of recognition by global-genome nucleotide excision repair accounts for the high mutagenicity and persistence of aristolactam-DNA adducts.
Sidorenko VS et al.
Nucleic Acids Research, 2012
Cockayne syndrome protein B interacts with and is phosphorylated by c-Abl tyrosine kinase.
Imam SZ et al.
Nucleic Acids Research, 2007
Differential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells.
Selzer RR et al.
Nucleic Acids Research, 2002
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