Associated Papers
The interaction of XPG with TFIIH through p62 and XPD is required for the completion of nucleotide excision repair.
Kim M et al.
Nucleic Acids Research, 2026
The interaction of XPG with TFIIH through p62 and XPD is required for the completion of nucleotide excision repair.
Kim M et al.
Biorxiv : the Preprint Server for Biology, 2025
Active DNA damage eviction by HLTF stimulates nucleotide excision repair.
van Toorn M et al.
Molecular Cell, 2022
XPF-ERCC1 protects liver, kidney and blood homeostasis outside the canonical excision repair pathways.
Mulderrig L et al.
Plos Genetics, 2020
Structural basis of TFIIH activation for nucleotide excision repair.
Kokic G et al.
Nature Communications, 2019
BERing the burden of damage: Pathway crosstalk and posttranslational modification of base excision repair proteins regulate DNA damage management.
Limpose KL et al.
Dna Repair, 2017
Transcription-coupled nucleotide excision repair factors promote R-loop-induced genome instability.
Sollier J et al.
Molecular Cell, 2014
Cell-autonomous progeroid changes in conditional mouse models for repair endonuclease XPG deficiency.
Barnhoorn S et al.
Plos Genetics, 2014
The PCNA binding domain of Rad2p plays a role in mutagenesis by modulating the cell cycle in response to DNA damage.
Yu SL et al.
Dna Repair, 2014
FANCJ localization by mismatch repair is vital to maintain genomic integrity after UV irradiation.
Guillemette S et al.
Cancer Research, 2014
XPG and XPF endonucleases trigger chromatin looping and DNA demethylation for accurate expression of activated genes.
Le May N et al.
Molecular Cell, 2012
XPG: its products and biological roles.
Schärer OD
Advances in Experimental Medicine and Biology, 2008
Tissue specific mutagenic and carcinogenic responses in NER defective mouse models.
Wijnhoven SW et al.
Mutation Research, 2007
Suppression of UV-induced apoptosis by the human DNA repair protein XPG.
Clément V et al.
Cell Death and Differentiation, 2006
The spacer region of XPG mediates recruitment to nucleotide excision repair complexes and determines substrate specificity.
Dunand-Sauthier I et al.
The Journal of Biological Chemistry, 2005
Identification of the XPG region that causes the onset of Cockayne syndrome by using Xpg mutant mice generated by the cDNA-mediated knock-in method.
Shiomi N et al.
Molecular and Cellular Biology, 2004
Deficiency in the nuclease activity of xeroderma pigmentosum G in mice leads to hypersensitivity to UV irradiation.
Tian M et al.
Molecular and Cellular Biology, 2004
Requirement of yeast RAD2, a homolog of human XPG gene, for efficient RNA polymerase II transcription. implications for Cockayne syndrome.
Lee SK et al.
Cell, 2002
TFIIH with inactive XPD helicase functions in transcription initiation but is defective in DNA repair.
Winkler GS et al.
The Journal of Biological Chemistry, 2000
Conserved residues of human XPG protein important for nuclease activity and function in nucleotide excision repair.
Constantinou A et al.
The Journal of Biological Chemistry, 1999
Base excision repair of oxidative DNA damage activated by XPG protein.
Klungland A et al.
Molecular Cell, 1999
Cockayne syndrome pathogenesis: lessons from mouse models.
Jaarsma D et al.
Mechanisms of Ageing and Development, 0