Associated Papers
Quantitative hypermorphic FAM111A alleles cause autosomal recessive Kenny-Caffey syndrome type 2 and osteocraniostenosis.
Li D et al.
Jci Insight, 2025
Dimerization-dependent serine protease activity of FAM111A prevents replication fork stalling at topoisomerase 1 cleavage complexes.
Palani S et al.
Nature Communications, 2024
Unravelling the Intricate Roles of FAM111A and FAM111B: From Protease-Mediated Cellular Processes to Disease Implications.
Naicker D et al.
International Journal of Molecular Sciences, 2024
FAM111A regulates replication origin activation and cell fitness.
Rios-Szwed DO et al.
Life Science Alliance, 2023
Functions and evolution of FAM111 serine proteases.
Welter AL et al.
Frontiers in Molecular Biosciences, 2022
FAM111A induces nuclear dysfunction in disease and viral restriction.
Nie M et al.
Embo Reports, 2021
FAM111A protects replication forks from protein obstacles via its trypsin-like domain.
Kojima Y et al.
Nature Communications, 2020
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