Associated Papers
[Mouse models for spontaneous dermatitis due to impaired skin barrier formation].
Sasaki T et al.
Nihon Rinsho Men'eki Gakkai Kaishi = Japanese Journal of Clinical Immunology, 2014
A homozygous nonsense mutation in the gene for Tmem79, a component for the lamellar granule secretory system, produces spontaneous eczema in an experimental model of atopic dermatitis.
Sasaki T et al.
The Journal of Allergy and Clinical Immunology, 2013
Tmem79/Matt is the matted mouse gene and is a predisposing gene for atopic dermatitis in human subjects.
Saunders SP et al.
The Journal of Allergy and Clinical Immunology, 2013
The human epidermal differentiation complex: cornified envelope precursors, S100 proteins and the 'fused genes' family.
Kypriotou M et al.
Experimental Dermatology, 2012
Loss-of-function mutations within the filaggrin gene and atopic dermatitis.
Kawasaki H et al.
Current Problems in Dermatology, 2011
Filaggrin deficiency confers a paracellular barrier abnormality that reduces inflammatory thresholds to irritants and haptens.
Scharschmidt TC et al.
The Journal of Allergy and Clinical Immunology, 2009
A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming.
Fallon PG et al.
Nature Genetics, 2009
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