Associated Papers
Genetic variation of FUT3 gene in the Han population from Northern China.
Zhou S et al.
Transfusion Medicine (oxford, England), 2024
Estimation of Lewis-negative alleles by high-resolution melting analysis of three tag SNPs of FUT3.
Soejima M et al.
Vox Sanguinis, 2022
Norovirus infection and HBGA host genetic susceptibility in a birth community-cohort, Rio de Janeiro, Brazil.
Cantelli CP et al.
Infection, Genetics and Evolution : Journal of Molecular Epidemiology and Evolutionary Genetics in Infectious Diseases, 2020
Systematic sequence analysis of the FUT3 gene identifies 11 novel alleles in the Sindhi and Punjabi populations from Pakistan.
Zhao M et al.
Scientific Reports, 2020
Genetic variation of FUT3 in Ghanaians, Caucasians, and Mongolians.
Soejima M et al.
Transfusion, 2009
The fucosyltransferase gene family: an amazing summary of the underlying mechanisms of gene evolution.
Javaud C et al.
Genetica, 2003
Lewis enzyme (alpha1-3/4 fucosyltransferase) polymorphisms do not explain the Lewis phenotype in the gastric mucosa of a Portuguese population.
Serpa J et al.
Journal of Human Genetics, 2003
Distribution of Lewis (FUT3)genotype and allele: frequencies in a biethnic United States population.
Cakir B et al.
Annals of Hematology, 2002
The polymorphisms of fucosyltransferases.
Koda Y et al.
Legal Medicine (tokyo, Japan), 2001
Genetic background of Lewis negative blood group phenotype and its association with atherosclerotic disease in the NHLBI family heart study.
Salomaa V et al.
Journal of Internal Medicine, 2000
Significance of each of three missense mutations, G484A, G667A, and G808A, present in an inactive allele of the human Lewis gene (FUT3) for alpha(1,3/1,4)fucosyltransferase inactivation.
Pang H et al.
Glycoconjugate Journal, 1998
Five novel missense mutations of the Lewis gene (FUT3) in African (Xhosa) and Caucasian populations in South Africa.
Pang H et al.
Human Genetics, 1998
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