Associated Papers
Genetic Variations of the FUT3 Gene in Le(a-b-) Individuals and Their Association with Lewis Antibody Responses.
Nathalang O et al.
Medical Sciences (basel, Switzerland), 2025
Correlation Of Fut2 And Fut3 Gene Polymorphisms With Inflammatory Bowel Disease In Guangxi Zhuang Population.
Lv X et al.
International Journal of General Medicine, 2025
Genetic variation of FUT3 gene in the Han population from Northern China.
Zhou S et al.
Transfusion Medicine (oxford, England), 2024
Fluorescence Melting Curve Analysis for Concurrent Genotyping of Three Tag SNPs in FUT3.
Soejima M et al.
Diagnostics (basel, Switzerland), 2022
ABO, secretor, and Lewis carbohydrate histo-blood groups are associated with autoimmune neutropenia of early childhood in Danish patients.
Kløve-Mogensen K et al.
Transfusion, 2022
Estimation of Lewis-negative alleles by high-resolution melting analysis of three tag SNPs of FUT3.
Soejima M et al.
Vox Sanguinis, 2022
Prediction of various blood group systems using Korean whole-genome sequencing data.
Hyun J et al.
Plos One, 2022
Divergence in phenotyping and genotyping analysis of the Lewis histo-blood group system.
Donta A et al.
Transfusion Medicine (oxford, England), 2021
Estimation of genetic variation in the Secretor and Lewis genes in Iranian hospitalized children.
Farahmand M et al.
Transfusion Clinique Et Biologique : Journal De La Societe Francaise De Transfusion Sanguine, 2021
FUT2, Secretor Status and FUT3 Polymorphisms of Children with Acute Diarrhea Infected with Rotavirus and Norovirus in Brazil.
Loureiro Tonini MA et al.
Viruses, 2020
Norovirus infection and HBGA host genetic susceptibility in a birth community-cohort, Rio de Janeiro, Brazil.
Cantelli CP et al.
Infection, Genetics and Evolution : Journal of Molecular Epidemiology and Evolutionary Genetics in Infectious Diseases, 2020
Systematic sequence analysis of the FUT3 gene identifies 11 novel alleles in the Sindhi and Punjabi populations from Pakistan.
Zhao M et al.
Scientific Reports, 2020
Genetic Manipulation of Human Intestinal Enteroids Demonstrates the Necessity of a Functional Fucosyltransferase 2 Gene for Secretor-Dependent Human Norovirus Infection.
Haga K et al.
Mbio, 2020
Associations between fucosyltransferase 3 gene polymorphisms and ankylosing spondylitis: A case-control study of an east Chinese population.
Jiang G et al.
Plos One, 2020
Genomic coordinates and continental distribution of 120 blood group variants reported by the 1000 Genomes Project.
Montemayor-Garcia C et al.
Transfusion, 2018
Distribution of Lewis and Secretor polymorphisms and corresponding CA19-9 antigen expression in a Chinese population.
Guo M et al.
Febs Open Bio, 2017
A common genetic variant of fucosyltransferase 2 correlates with serum carcinoembryonic antigen levels and affects cancer screening in patients with primary sclerosing cholangitis.
Wannhoff A et al.
United European Gastroenterology Journal, 2016
Association of elevated rotavirus-specific antibody titers with HBGA secretor status in Swedish individuals: The FUT2 gene as a putative susceptibility determinant for infection.
Günaydın G et al.
Virus Research, 2016
The Blood Group A Genotype Determines the Level of Expression of the Blood Group A on Platelets But Not the Anti-B Isotiter.
Lehner B et al.
Transfusion Medicine and Hemotherapy : Offizielles Organ Der Deutschen Gesellschaft Fur Transfusionsmedizin Und Immunhamatologie, 2015
Lewis histo-blood group system phenotyping and genotyping reveal divergence in the association of Le(a-b-) phenotype and type 1 diabetes.
Previato M et al.
Vox Sanguinis, 2015
Investigation of unexpected serum CA19-9 elevation in Lewis-negative cancer patients.
Hamada E et al.
Annals of Clinical Biochemistry, 2012
Polymorphisms of Lewis and Secretor genes are related to breast cancer and metastasis in axillary lymph nodes.
Teresa DB et al.
Tumour Biology : the Journal of the International Society for Oncodevelopmental Biology and Medicine, 2010
Identification of 14 new alleles at the fucosyltransferase 1, 2, and 3 loci in Styrian blood donors, Austria.
Matzhold EM et al.
Transfusion, 2009
Genetic variation of FUT3 in Ghanaians, Caucasians, and Mongolians.
Soejima M et al.
Transfusion, 2009
The G428A nonsense mutation in FUT2 provides strong but not absolute protection against symptomatic GII.4 Norovirus infection.
Carlsson B et al.
Plos One, 2009
Fucosyltransferase 3 polymorphism and atherothrombotic disease in the Framingham Offspring Study.
Djoussé L et al.
American Heart Journal, 2007
[FUT3 gene polymorphism associated with Lewis blood group in Chinese Zhejiang population].
Xu XG et al.
Zhongguo Shi Yan Xue Ye Xue Za Zhi, 2006
Association of the Lewis genotype with cardiovascular risk factors and subclinical carotid atherosclerosis: the Atherosclerosis Risk in Communities (ARIC) study.
Cakir B et al.
Journal of Internal Medicine, 2004
The fucosyltransferase gene family: an amazing summary of the underlying mechanisms of gene evolution.
Javaud C et al.
Genetica, 2003
Lewis enzyme (alpha1-3/4 fucosyltransferase) polymorphisms do not explain the Lewis phenotype in the gastric mucosa of a Portuguese population.
Serpa J et al.
Journal of Human Genetics, 2003
Distribution of Lewis (FUT3)genotype and allele: frequencies in a biethnic United States population.
Cakir B et al.
Annals of Hematology, 2002
Genotype at the secretor blood group locus is a determinant of plasma von Willebrand factor level.
O'Donnell J et al.
British Journal of Haematology, 2002
Identification of two functionally deficient plasma alpha 3-fucosyltransferase (FUT6) alleles.
Elmgren A et al.
Human Mutation, 2000
Lewis (FUT3) genotypes in Taiwanese, Thai, and Filipino populations.
Liu TC et al.
Annals of Hematology, 2000
Genetic background of Lewis negative blood group phenotype and its association with atherosclerotic disease in the NHLBI family heart study.
Salomaa V et al.
Journal of Internal Medicine, 2000
Molecular behavior of mutant Lewis enzymes in vivo.
Nishihara S et al.
Glycobiology, 1999
Typing for the human lewis blood group system by quantitative fluorescence-activated flow cytometry: large differences in antigen presentation on erythrocytes between A(1), A(2), B, O phenotypes.
Larson G et al.
Vox Sanguinis, 1999
Significance of each of three missense mutations, G484A, G667A, and G808A, present in an inactive allele of the human Lewis gene (FUT3) for alpha(1,3/1,4)fucosyltransferase inactivation.
Pang H et al.
Glycoconjugate Journal, 1998
Five novel missense mutations of the Lewis gene (FUT3) in African (Xhosa) and Caucasian populations in South Africa.
Pang H et al.
Human Genetics, 1998
Conserved structural features in eukaryotic and prokaryotic fucosyltransferases.
Breton C et al.
Glycobiology, 1998
Point mutations and deletion responsible for the Bombay H null and the Reunion H weak blood groups.
Fernandez-Mateos P et al.
Vox Sanguinis, 1998
Significance of individual point mutations, T202C and C314T, in the human Lewis (FUT3) gene for expression of Lewis antigens by the human alpha(1,3/1,4)-fucosyltransferase, Fuc-TIII.
Elmgren A et al.
The Journal of Biological Chemistry, 1997
Influence of Lewis alpha1-3/4-L-fucosyltransferase (FUT3) gene mutations on enzyme activity, erythrocyte phenotyping, and circulating tumor marker sialyl-Lewis a levels.
Orntoft TF et al.
The Journal of Biological Chemistry, 1996
Correlation of FUT3 and FUT6 Gene Polymorphisms With Helicobacter pylori Infection.
Zhou S et al.
Helicobacter, 0