Associated Papers
Increased SLC38A4 Amino Acid Transporter Expression in Human Pancreatic α-Cells After Glucagon Receptor Inhibition.
Kim J et al.
Endocrinology, 2019
The first pediatric case of glucagon receptor defect due to biallelic mutations in GCGR is identified by newborn screening of elevated arginine.
Li H et al.
Molecular Genetics and Metabolism Reports, 2018
Hypercalcemia in Glucagon Cell Hyperplasia and Neoplasia (Mahvash Syndrome): A New Association.
Gild ML et al.
The Journal of Clinical Endocrinology and Metabolism, 2018
Glucagon cell hyperplasia and neoplasia with and without glucagon receptor mutations.
Sipos B et al.
The Journal of Clinical Endocrinology and Metabolism, 2015
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