Associated Papers
Exploring the Potent Roles of an Internally Translated Truncated Connexin-43 Isoform.
Maalouf M et al.
Biology, 2024
Connexin Mutations and Hereditary Diseases.
Qiu Y et al.
International Journal of Molecular Sciences, 2022
Removal of an Internal Translational Start Site from mRNA While Retaining Expression of the Full-Length Protein.
Shimura D et al.
Journal of Visualized Experiments : Jove, 2022
Caveolin-3 and Arrhythmias: Insights into the Molecular Mechanisms.
He M et al.
Journal of Clinical Medicine, 2022
Internal translation of Gja1 (Connexin43) to produce GJA1-20k: Implications for arrhythmia and ischemic-preconditioning.
Whisenant CC et al.
Frontiers in Physiology, 2022
Remodeling of Ion Channel Trafficking and Cardiac Arrhythmias.
Blandin CE et al.
Cells, 2021
Auxiliary trafficking subunit GJA1-20k protects connexin-43 from degradation and limits ventricular arrhythmias.
Xiao S et al.
The Journal of Clinical Investigation, 2020
Gap junction protein Connexin-43 is a direct transcriptional regulator of N-cadherin in vivo.
Kotini M et al.
Nature Communications, 2018
Autoregulation of connexin43 gap junction formation by internally translated isoforms.
Smyth JW et al.
Cell Reports, 2013
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