Associated Papers
Dysbiosis and Neurodegeneration in ALS: Unraveling the Gut-Brain Axis.
Sharma VK
Neuromolecular Medicine, 2025
5-Hydroxytryptamine: a potential therapeutic target in amyotrophic lateral sclerosis.
Jiang SS et al.
Neural Regeneration Research, 2023
cGAS and DDX41-STING mediated intrinsic immunity spreads intercellularly to promote neuroinflammation in SOD1 ALS model.
Tan HY et al.
Iscience, 2022
Amyotrophic Lateral Sclerosis Genes in Drosophila melanogaster.
Layalle S et al.
International Journal of Molecular Sciences, 2021
Genetic approach in amyotrophic lateral sclerosis.
Cervantes-Aragón I et al.
Gaceta Medica De Mexico, 2019
Sigma 1 receptor activation modifies intracellular calcium exchange in the G93AhSOD1 ALS model.
Tadić V et al.
Neuroscience, 2017
Are Astrocytes the Predominant Cell Type for Activation of Nrf2 in Aging and Neurodegeneration?
Liddell JR
Antioxidants (basel, Switzerland), 2017
Altered nucleocytoplasmic proteome and transcriptome distributions in an in vitro model of amyotrophic lateral sclerosis.
Kim JE et al.
Plos One, 2017
Up-regulation of neural and cell cycle-related microRNAs in brain of amyotrophic lateral sclerosis mice at late disease stage.
Marcuzzo S et al.
Molecular Brain, 2015
Expression of wild-type human superoxide dismutase-1 in mice causes amyotrophic lateral sclerosis.
Graffmo KS et al.
Human Molecular Genetics, 2013
Nuclear localization of human SOD1 and mutant SOD1-specific disruption of survival motor neuron protein complex in transgenic amyotrophic lateral sclerosis mice.
Gertz B et al.
Journal of Neuropathology and Experimental Neurology, 2012
Nrf2/ARE Signaling Pathway: Key Mediator in Oxidative Stress and Potential Therapeutic Target in ALS.
Petri S et al.
Neurology Research International, 2012
The Role of immune and inflammatory mechanisms in ALS.
McCombe PA et al.
Current Molecular Medicine, 2011
Genetic rodent models of amyotrophic lateral sclerosis.
Van Den Bosch L
Journal of Biomedicine & Biotechnology, 2011
Neuron-specific expression of mutant superoxide dismutase is sufficient to induce amyotrophic lateral sclerosis in transgenic mice.
Jaarsma D et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 2008