Associated Papers
Mechanisms and Physiological Roles of Polymorphisms in Gestational Diabetes Mellitus.
Suthon S et al.
International Journal of Molecular Sciences, 2024
Molecular dynamics simulations and bioinformatics' analysis of deleterious missense single nucleotide polymorphisms in Glyoxalase-1 gene.
Syed NA et al.
Journal of Biomolecular Structure & Dynamics, 2023
Association between GLO1 variants and gestational diabetes mellitus susceptibility in a Chinese population: a preliminary study.
Zeng Q et al.
Frontiers in Endocrinology, 2023
Sensitization of Patient-Derived Colorectal Cancer Organoids to Photon and Proton Radiation by Targeting DNA Damage Response Mechanisms.
Pape K et al.
Cancers, 2022
Genetic Variants Associated with Chronic Kidney Disease in a Spanish Population.
Corredor Z et al.
Scientific Reports, 2020
Common variants in glyoxalase I do not increase chronic pancreatitis risk.
Kaune T et al.
Plos One, 2019
GLO1 gene polymorphisms and their association with retinitis pigmentosa: a case-control study in a Sicilian population.
Donato L et al.
Molecular Biology Reports, 2018
Whole exome sequencing in three families segregating a pediatric case of sarcoidosis.
Calender A et al.
Bmc Medical Genomics, 2018
Renal oncocytoma characterized by the defective complex I of the respiratory chain boosts the synthesis of the ROS scavenger glutathione.
Kürschner G et al.
Oncotarget, 2017
The influence of glyoxalase 1 gene polymorphism on its expression at different stages of breast cancer in Egyptian women.
Abdul-Maksoud RS et al.
Genes & Cancer, 2017
Analysis of functional GLO1 variants in the BTBD9 locus and restless legs syndrome.
Gan-Or Z et al.
Sleep Medicine, 2015
Polymorphisms of the receptor for advanced glycation end-products and glyoxalase I in patients with renal cancer.
Chocholatý M et al.
Tumour Biology : the Journal of the International Society for Oncodevelopmental Biology and Medicine, 2015
The eSNV-detect: a computational system to identify expressed single nucleotide variants from transcriptome sequencing data.
Tang X et al.
Nucleic Acids Research, 2014
The GLO1 C332 (Ala111) allele confers autism vulnerability: family-based genetic association and functional correlates.
Gabriele S et al.
Journal of Psychiatric Research, 2014
Fructosamine 3-kinase and glyoxalase I polymorphisms and their association with soluble RAGE and adhesion molecules in diabetes.
Škrha J et al.
Physiological Research, 2014
Glyoxalase 1 enzyme activity in erythrocytes and Ala111Glu polymorphism in type 1-diabetes patients.
Sakhi AK et al.
Scandinavian Journal of Clinical and Laboratory Investigation, 2013
Receptor for advanced glycation end products (RAGE) and glyoxalase I gene polymorphisms in pathological pregnancy.
Germanová A et al.
Clinical Biochemistry, 2012
Glyoxalase I and aldose reductase gene polymorphisms and susceptibility to carotid atherosclerosis in type 2 diabetes.
Wu JC et al.
Genetic Testing and Molecular Biomarkers, 2011
[Studies on pathophysiology of schizophrenia with a rare variant as a clue].
Itokawa M et al.
Brain and Nerve = Shinkei Kenkyu No Shinpo, 2011
[Molecular pathophysiology of schizophrenia and preventive strategy in pubertal period].
Itokawa M et al.
Seishin Shinkeigaku Zasshi = Psychiatria Et Neurologia Japonica, 2011
Glyoxalase I Ala111Glu gene polymorphism: No association with breast cancer risk but correlated with absence of progesterone receptor.
Naidu R et al.
Pathology International, 2010
Glyoxalase I Glu111Ala polymorphism in patients with breast cancer.
Germanová A et al.
Cancer Investigation, 2009
A419C (E111A) polymorphism of the glyoxalase I gene and vascular complications in chronic hemodialysis patients.
Kalousová M et al.
Annals of the New York Academy of Sciences, 2008
Role for glyoxalase I in Alzheimer's disease.
Chen F et al.
Proceedings of the National Academy of Sciences of the United States of America, 2004
Determination of Glyoxalase-1 levels and Identification of Genetic Variants in GLO1 Gene in Patients of Diabetic Nephropathy.
Shah SZH et al.
Pakistan Journal of Medical Sciences, 0