Associated Papers
Metabolic gene polymorphisms and type 2 diabetes mellitus risk: A systematic review and meta-analyses.
Indarto D et al.
Experimental and Therapeutic Medicine, 2026
GWAS of random glucose in 476,326 individuals provide insights into diabetes pathophysiology, complications and treatment stratification.
Lagou V et al.
Nature Genetics, 2023
37679419 (PubMed )
Supplement: 41588_2023_1462_MOESM6_ESM.xlsx › SuppTable11
Novel NOG (p.P42S) mutation causes proximal symphalangism in a four-generation Chinese family.
Sha Y et al.
Bmc Medical Genetics, 2019
31370824 (PubMed )
Supplement: 12881_2019_864_MOESM2_ESM.xlsx › Sheet1
Association of genetic variants of the incretin-related genes with quantitative traits and occurrence of type 2 diabetes in Japanese.
Enya M et al.
Molecular Genetics and Metabolism Reports, 2014
Minireview: Signal bias, allosterism, and polymorphic variation at the GLP-1R: implications for drug discovery.
Koole C et al.
Molecular Endocrinology (baltimore, Md.), 2013
Pharmacological characterization of human incretin receptor missense variants.
Fortin JP et al.
The Journal of Pharmacology and Experimental Therapeutics, 2010
Five missense mutations in glucagon-like peptide 1 receptor gene in Japanese population.
Tokuyama Y et al.
Diabetes Research and Clinical Practice, 2004
A small molecule ligand of the glucagon-like peptide 1 receptor targets its amino-terminal hormone binding domain.
Tibaduiza EC et al.
The Journal of Biological Chemistry, 2001
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