Associated Papers
Somatic mutations and outcomes in chronic myeloid leukemia adolescent and young adults compared to children, adults, and BCR::ABL1-positive acute lymphoblastic leukemia.
Krizkova J et al.
Leukemia, 2025
40295826 (PubMed )
Supplement: 41375_2025_2609_MOESM1_ESM.pdf
Mutation and cell state compatibility is required and targetable in Ph+ acute lymphoblastic leukemia minimal residual disease.
Winter PS et al.
Biorxiv : the Preprint Server for Biology, 2024
38915726 (PubMed )
Supplement: media-2.pdf
Clinical characterization of familial 1p36.3 microduplication.
Jiao J et al.
Neurogenetics, 2023
Genome-wide analyses of 200,453 individuals yield new insights into the causes and consequences of clonal hematopoiesis.
Kar SP et al.
Nature Genetics, 2022
35835912 (PubMed )
Supplement: 41588_2022_1121_MOESM3_ESM.xlsx › ST2
The Emerging Role of Gβ Subunits in Human Genetic Diseases.
Malerba N et al.
Cells, 2019
An activating mutation of GNB1 is associated with resistance to tyrosine kinase inhibitors in ETV6-ABL1-positive leukemia.
Zimmermannova O et al.
Oncogene, 2017
Acute lymphoblastic leukemia in a child with a de novo germline gnb1 mutation.
Brett M et al.
American Journal of Medical Genetics. Part A, 2017
Pooled screening for antiproliferative inhibitors of protein-protein interactions.
Nim S et al.
Nature Chemical Biology, 2016
Mutations in G protein β subunits promote transformation and kinase inhibitor resistance.
Yoda A et al.
Nature Medicine, 2015
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