Associated Papers
Mutations remote from the human gonadotropin-releasing hormone (GnRH) receptor-binding sites specifically increase binding affinity for GnRH II but not GnRH I: evidence for ligand-selective, receptor-active conformations.
Lu ZL et al.
The Journal of Biological Chemistry, 2005
A single amino acid substitution in transmembrane helix VI results in overexpression of the human GnRH receptor.
Myburgh DB et al.
European Journal of Endocrinology, 1998
Select a paper to view details