Associated Papers
Nanobodies restore stability to cancer-associated mutants of tumor suppressor protein p16INK4a.
Burbidge O et al.
Structure (london, England : 1993), 2025
Analysis and Interpretation of the Impact of Missense Variants in Cancer.
Petrosino M et al.
International Journal of Molecular Sciences, 2021
34063805 (PubMed )
Supplement: ijms-22-05416-s001.zip › new_supplementary_file1.xlsx › new_supplementary_file1
p16 mutation spectrum in the premalignant condition Barrett's esophagus.
Paulson TG et al.
Plos One, 2008
Genetic and epigenetic inactivation of LTF gene at 3p21.3 in lung cancers.
Iijima H et al.
International Journal of Cancer, 2006
The ankyrin repeat as molecular architecture for protein recognition.
Mosavi LK et al.
Protein Science : a Publication of the Protein Society, 2004
Detailed computational study of p53 and p16: using evolutionary sequence analysis and disease-associated mutations to predict the functional consequences of allelic variants.
Greenblatt MS et al.
Oncogene, 2003
Biologic and biochemical analyses of p16(INK4a) mutations from primary tumors.
Yarbrough WG et al.
Journal of the National Cancer Institute, 1999
Stability and folding of the tumour suppressor protein p16.
Tang KS et al.
Journal of Molecular Biology, 1999
Defective folding of mutant p16(INK4) proteins encoded by tumor-derived alleles.
Zhang B et al.
The Journal of Biological Chemistry, 1996
Select a paper to view details