Associated Papers
TDP-43 protein interactome informs about perturbed canonical pathways and may help develop personalized medicine approaches for patients with TDP-43 pathology.
Helmold BR et al.
Drug Discovery Today, 2023
Intracellular Conformation of Amyotrophic Lateral Sclerosis-Causative TDP-43.
Kitamura A et al.
International Journal of Molecular Sciences, 2023
Specific RNA interactions promote TDP-43 multivalent phase separation and maintain liquid properties.
Grese ZR et al.
Embo Reports, 2021
Amyotrophic Lateral Sclerosis Genes in Drosophila melanogaster.
Layalle S et al.
International Journal of Molecular Sciences, 2021
A Systematic and Comprehensive Review on Disease-Causing Genes in Amyotrophic Lateral Sclerosis.
Srinivasan E et al.
Journal of Molecular Neuroscience : Mn, 2020
The debated toxic role of aggregated TDP-43 in amyotrophic lateral sclerosis: a resolution in sight?
Hergesheimer RC et al.
Brain : a Journal of Neurology, 2019
From animal models to human disease: a genetic approach for personalized medicine in ALS.
Picher-Martel V et al.
Acta Neuropathologica Communications, 2016
Clinical and genetic basis of familial amyotrophic lateral sclerosis.
Souza PV et al.
Arquivos De Neuro-psiquiatria, 2015
ALS-linked mutations enlarge TDP-43-enriched neuronal RNA granules in the dendritic arbor.
Liu-Yesucevitz L et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 2014
Global analysis of TDP-43 interacting proteins reveals strong association with RNA splicing and translation machinery.
Freibaum BD et al.
Journal of Proteome Research, 2010
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