Associated Papers
A common form of dominant human IFNAR1 deficiency impairs IFN-α and -ω but not IFN-β-dependent immunity.
Al Qureshah F et al.
The Journal of Experimental Medicine, 2025
Participation of Single-Nucleotide Variants in IFNAR1 and IFNAR2 in the Immune Response against SARS-CoV-2 Infection: A Systematic Review.
López-Bielma MF et al.
Pathogens (basel, Switzerland), 2023
Individual genetic variability mainly of Proinflammatory cytokines, cytokine receptors, and toll-like receptors dictates pathophysiology of COVID-19 disease.
Vakil MK et al.
Journal of Medical Virology, 2022
The association of COVID-19 severity and susceptibility and genetic risk factors: A systematic review of the literature.
Ishak A et al.
Gene, 2022
Null IFNAR1 and IFNAR2 alleles are surprisingly common in the Pacific and Arctic.
Meyts I
The Journal of Experimental Medicine, 2022
Inherited IFNAR1 Deficiency in a Child with Both Critical COVID-19 Pneumonia and Multisystem Inflammatory Syndrome.
Abolhassani H et al.
Journal of Clinical Immunology, 2022
An overview of human proteins and genes involved in SARS-CoV-2 infection.
Jahanafrooz Z et al.
Gene, 2022
Human genetic basis of severe or critical illness in COVID-19.
Ji XS et al.
Frontiers in Cellular and Infection Microbiology, 2022
Loss-of-function mutations in IFNAR2 in COVID-19 severe infection susceptibility.
Smieszek SP et al.
Journal of Global Antimicrobial Resistance, 2021
Inborn errors of type I IFN immunity in patients with life-threatening COVID-19.
Zhang Q et al.
Science (new York, N.y.), 2020
Select a paper to view details