Varaico: Literature-Extracted Genetic Variants

Gene:IL2RA
Variant:p.T106T
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IL2RA

NM_000417.3:p.T106T

Associated Papers

-318C/T polymorphism of the CTLA-4 gene is an independent risk factor for RBC alloimmunization among sickle cell disease patients.

Oliveira VB et al.

International Journal of Immunogenetics, 2017

Association of CTLA-4 gene polymorphisms -318C/T and +49A/G and Hashimoto's thyroidits in Zahedan, Iran.

Narooie-Nejad M et al.

Biomedical Reports, 2017

The influence of CTLA-4 single nucleotide polymorphisms on acute kidney allograft rejection in Turkish patients.

Ruhi Ç et al.

Clinical Transplantation, 2015

Association of CTLA-4 variants with susceptibility to inflammatory bowel disease: a meta-analysis.

Zhang M et al.

Human Immunology, 2014

CTLA-4 exon 1 +49 polymorphism alone and in a haplotype with -318 promoter polymorphism may confer susceptibility to chronic HBV infection in Chinese Han patients.

Duan S et al.

Molecular Biology Reports, 2011

Association of single nucleotide polymorphisms in cytotoxic T-lymphocyte antigen 4 and susceptibility to autoimmune type 1 diabetes in Tunisians.

Benmansour J et al.

Clinical and Vaccine Immunology : Cvi, 2010

CTLA4 gene variations and haplotypes in patients with lung cancer.

Khaghanzadeh N et al.

Cancer Genetics and Cytogenetics, 2010

CTLA-4 single-nucleotide polymorphisms in a Caucasian population with schizophrenia.

Jones AL et al.

Brain, Behavior, and Immunity, 2009

Association of -318 C/T and +49 A/G cytotoxic T lymphocyte antigen-4 (CTLA-4) gene polymorphisms with a clinical subset of Italian patients with systemic sclerosis.

Balbi G et al.

Clinical and Experimental Immunology, 2007

Cytotoxic T lymphocyte antigen-4 promoter variants in breast cancer.

Erfani N et al.

Cancer Genetics and Cytogenetics, 2006

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Regarding the variants extracted from papers and shown text snippets from research papers, the following holds: "Some rights reserved. This work permits non-commercial use, distribution and reproduction in any medium, provided the original author and source are credited."