Associated Papers
Using Zebrafish Animal Model to Study the Genetic Underpinning and Mechanism of Arrhythmogenic Cardiomyopathy.
Niu Y et al.
International Journal of Molecular Sciences, 2023
Genetic Animal Models for Arrhythmogenic Cardiomyopathy.
Gerull B et al.
Frontiers in Physiology, 2020
Therapeutic Chemical Screen Identifies Phosphatase Inhibitors to Reconstitute PKB Phosphorylation and Cardiac Contractility in ILK-Deficient Zebrafish.
Pott A et al.
Biomolecules, 2018
Genetics and disease of ventricular muscle.
Fatkin D et al.
Cold Spring Harbor Perspectives in Medicine, 2014
Biochemical, proteomic, structural, and thermodynamic characterizations of integrin-linked kinase (ILK): cross-validation of the pseudokinase.
Fukuda K et al.
The Journal of Biological Chemistry, 2011
Affixin (β-parvin) promotes cardioprotective signaling via STAT3 activation.
Luedde M et al.
Journal of Molecular and Cellular Cardiology, 2011
Laminin-alpha4 and integrin-linked kinase mutations cause human cardiomyopathy via simultaneous defects in cardiomyocytes and endothelial cells.
Knöll R et al.
Circulation, 2007
Paxillin and ponsin interact in nascent costameres of muscle cells.
Gehmlich K et al.
Journal of Molecular Biology, 2007
Integrin-linked kinase, a novel component of the cardiac mechanical stretch sensor, controls contractility in the zebrafish heart.
Bendig G et al.
Genes & Development, 2006
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