Associated Papers
ATR1 A1166C (rs5186), FII G20210A (rs1799963), FV G1691A (rs6025), FXIII 97G > T (rs11466016) and MTHFR A1298C (rs1801131) polymorphisms and the risk of ST-elevation myocardial infarction in young Mexican individuals.
Isordia-Salas I et al.
Molecular Biology Reports, 2024
Impact of pharmacogenetics on aspirin resistance: a systematic review.
Silva GFD et al.
Arquivos De Neuro-psiquiatria, 2023
The Association of Hereditary Prothrombotic Risk Factors with ST-Elevation Myocardial Infarction.
Damar İH et al.
Medeniyet Medical Journal, 2020
Hemostasis-related gene polymorphisms and their epistatic relationship in women with idiopathic infertility.
Velickovic J et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis, 2019
Glanzmann Thrombasthenia in a Newborn with Heterozygous Factor V Leiden and Heterozygous MTHFR C677T Gene Mutations.
Gultekin ND et al.
Indian Pediatrics, 2019
Genetics of suspected thrombophilia in Serbian females with infertility, including three cases, homozygous for FII 20210A or FV 1691A mutations.
Djurovic J et al.
Human Fertility (cambridge, England), 2017
Atherosclerotic and thrombotic genetic and environmental determinants in Egyptian coronary artery disease patients: a pilot study.
Fawzy MS et al.
Bmc Cardiovascular Disorders, 2017
Genetic polymorphisms, Biochemical Factors, and Conventional Risk Factors in Young and Elderly North Indian Patients With Acute Myocardial Infarction.
Kaur R et al.
Clinical and Applied Thrombosis/hemostasis : Official Journal of the International Academy of Clinical and Applied Thrombosis/hemostasis, 2016
Prothrombotic gene polymorphisms and plasma factors in young North Indian survivors of acute myocardial infarction.
Dogra RK et al.
Journal of Thrombosis and Thrombolysis, 2012
Pharmacogenomics in thrombosis.
Mousa SA
Methods in Molecular Biology (clifton, N.j.), 2010
Impact of hemostatic gene single point mutations in patients with non-diabetic coronary artery disease.
Var A et al.
Molecular Biology Reports, 2009
The factor V G1691A, factor V H1299R, prothrombin G20210A polymorphisms in children with family history of premature coronary artery disease.
Ciftdoğan DY et al.
Coronary Artery Disease, 2009
[Clinicomorphological characteristics of renal disorders in patients with genetic thrombophilia].
Kozlovskaia NL et al.
Terapevticheskii Arkhiv, 2009
[Factor V Leiden and C677T MTHFR thrombophilic mutations associated with hyperfibrinogenemia, persistent high CRP and leucocytosis in newborn baby with perinatal infection--a clinical case].
Vulkova A et al.
Akusherstvo I Ginekologiia, 2009
Genetic and acquired prothrombotic risk factors and sudden hearing loss.
Capaccio P et al.
The Laryngoscope, 2007
Association of PLA2 polymorphism of the ITGB3 gene with early fetal loss.
Ruzzi L et al.
Fertility and Sterility, 2005
Ramifications of four concurrent thrombophilic mutations and one hypofibrinolytic mutation.
Glueck CJ et al.
Clinical and Applied Thrombosis/hemostasis : Official Journal of the International Academy of Clinical and Applied Thrombosis/hemostasis, 2004
Platelet GPIaC807T polymorphism is associated with negative outcome of sudden hearing loss.
Rudack C et al.
Hearing Research, 2004
Combined thrombophilic polymorphisms in women with idiopathic recurrent miscarriage.
Hohlagschwandtner M et al.
Fertility and Sterility, 2003
[Gene polymorphisms of hemostasis and coronary risk].
Hellstern P et al.
Medizinische Klinik (munich, Germany : 1983), 2001
Prothrombotic genetic risk factors in young survivors of myocardial infarction.
Ardissino D et al.
Blood, 1999
Thrombophilic DNA Mutations As Independent Risk Factors for Stroke and Avascular Necrosis in Sickle Cell Anemia.
Zimmerman SA et al.
Hematology (amsterdam, Netherlands), 0
[Role of polymorphic markers for the genes of hemostasis and platelet receptors in liver fibrosis progression in patients with chronic hepatitis C].
Starostina EE et al.
Terapevticheskii Arkhiv, 0
Molecular assessment of some cardiovascular genetic risk factors among Iraqi patients with ischemic heart diseases.
Mohammed WJ et al.
International Journal of Health Sciences, 0