Associated Papers
A mutation in KCNJ11 causing human hyperinsulinism (Y12X) results in a glucose-intolerant phenotype in the mouse.
Hugill A et al.
Diabetologia, 2010
K(ATP) channelopathies in the pancreas.
Remedi MS et al.
Pflugers Archiv : European Journal of Physiology, 2010
ATP-sensitive potassium channels in health and disease.
Clark R et al.
Advances in Experimental Medicine and Biology, 2010
Hyperinsulinism and diabetes: genetic dissection of beta cell metabolism-excitation coupling in mice.
Remedi MS et al.
Cell Metabolism, 2009
Subunit-stoichiometric evidence for kir6.2 channel gating, ATP binding, and binding-gating coupling.
Wang R et al.
Molecular Pharmacology, 2007
Hyperinsulinism in mice with heterozygous loss of K(ATP) channels.
Remedi MS et al.
Diabetologia, 2006
Hyperinsulinism in infancy: from basic science to clinical disease.
Dunne MJ et al.
Physiological Reviews, 2004
Diabetes and insulin secretion: whither KATP?
Nichols CG et al.
American Journal of Physiology. Endocrinology and Metabolism, 2002
K(ATP) channels and insulin secretion disorders.
Huopio H et al.
American Journal of Physiology. Endocrinology and Metabolism, 2002
Potassium channels: molecular defects, diseases, and therapeutic opportunities.
Shieh CC et al.
Pharmacological Reviews, 2000
The weaver mouse gain-of-function phenotype of dopaminergic midbrain neurons is determined by coactivation of wvGirk2 and K-ATP channels.
Liss B et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 1999
Molecular biology of adenosine triphosphate-sensitive potassium channels.
Aguilar-Bryan L et al.
Endocrine Reviews, 1999
Subunit stoichiometry of the pancreatic beta-cell ATP-sensitive K+ channel.
Inagaki N et al.
Febs Letters, 1997
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