Associated Papers
Genetics of migraine: where are we now?
Grangeon L et al.
The Journal of Headache and Pain, 2023
Pathophysiology of migraine aura.
Sanchez Del Rio M et al.
Handbook of Clinical Neurology, 2023
Regulation of two-pore-domain potassium TREK channels and their involvement in pain perception and migraine.
Ávalos Prado P et al.
Neuroscience Letters, 2022
A review of migraine genetics: gathering genomic and transcriptomic factors.
Dias A et al.
Human Genetics, 2022
K2P18.1 translates T cell receptor signals into thymic regulatory T cell development.
Ruck T et al.
Cell Research, 2022
Pathophysiological Role of K2P Channels in Human Diseases.
Lee LM et al.
Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology, 2021
Comparison of K+ Channel Families.
Taura J et al.
Handbook of Experimental Pharmacology, 2021
TRESK and TREK-2 two-pore-domain potassium channel subunits form functional heterodimers in primary somatosensory neurons.
Lengyel M et al.
The Journal of Biological Chemistry, 2020
The Background K+ Channel TRESK in Sensory Physiology and Pain.
Andres-Bilbe A et al.
International Journal of Molecular Sciences, 2020
Genetics of migraine aura: an update.
de Boer I et al.
The Journal of Headache and Pain, 2020
Analysis of Genetic Variants in SCN1A, SCN2A, KCNK18, TRPA1 and STX1A as a Possible Marker of Migraine.
Kowalska M et al.
Current Genomics, 2020
A causal role for TRESK loss of function in migraine mechanisms.
Pettingill P et al.
Brain : a Journal of Neurology, 2019
Advances in genetics of migraine.
Sutherland HG et al.
The Journal of Headache and Pain, 2019
Migraine-Associated TRESK Mutations Increase Neuronal Excitability through Alternative Translation Initiation and Inhibition of TREK.
Royal P et al.
Neuron, 2019
[Two-pore-domain potassium channels and molecular mechanisms underlying migraine].
Royal P et al.
Biologie Aujourd'hui, 2019
Ion channelopathies and migraine pathogenesis.
Albury CL et al.
Molecular Genetics and Genomics : Mgg, 2017
Genetics of Migraine: Insights into the Molecular Basis of Migraine Disorders.
Sutherland HG et al.
Headache, 2017
Properties, regulation, pharmacology, and functions of the K₂p channel, TRESK.
Enyedi P et al.
Pflugers Archiv : European Journal of Physiology, 2015
Two-pore domain potassium channels: potential therapeutic targets for the treatment of pain.
Mathie A et al.
Pflugers Archiv : European Journal of Physiology, 2015
KCNK18 (TRESK) genetic variants in Italian patients with migraine.
Rainero I et al.
Headache, 2014
Nonmigraine-associated TRESK K+ channel variant C110R does not increase the excitability of trigeminal ganglion neurons.
Guo Z et al.
Journal of Neurophysiology, 2014
The molecular pathogenesis of migraine: new developments and opportunities.
Zameel Cader M
Human Molecular Genetics, 2013
Analysis of 3 common polymorphisms in the KCNK18 gene in an Australian Migraine case-control cohort.
Maher BH et al.
Gene, 2013
Pearls and pitfalls in genetic studies of migraine.
Eising E et al.
Cephalalgia : an International Journal of Headache, 2013
Functional analysis of missense variants in the TRESK (KCNK18) K channel.
Andres-Enguix I et al.
Scientific Reports, 2012
A dominant-negative mutation in the TRESK potassium channel is linked to familial migraine with aura.
Lafrenière RG et al.
Nature Medicine, 2010
[Ion channels and mechanisms of inherited disease transmission causing migraine].
Royal P et al.
Medecine Sciences : M/s, 0