Associated Papers
Dysregulation of alternative splicing underlies synaptic defects in familial amyotrophic lateral sclerosis.
Verdile V et al.
Progress in Neurobiology, 2023
SAM68 interaction with U1A modulates U1 snRNP recruitment and regulates mTor pre-mRNA splicing.
Subramania S et al.
Nucleic Acids Research, 2019
The RNA-binding protein Sam68 regulates expression and transcription function of the androgen receptor splice variant AR-V7.
Stockley J et al.
Scientific Reports, 2015
The transcription factor FBI-1 inhibits SAM68-mediated BCL-X alternative splicing and apoptosis.
Bielli P et al.
Embo Reports, 2014
The tissue-specific RNA binding protein T-STAR controls regional splicing patterns of neurexin pre-mRNAs in the brain.
Ehrmann I et al.
Plos Genetics, 2013
Spinal muscular atrophy: a new player joins the battle for SMN2 exon 7 splicing.
Pedrotti S et al.
Cell Cycle (georgetown, Tex.), 2010
The splicing regulator Sam68 binds to a novel exonic splicing silencer and functions in SMN2 alternative splicing in spinal muscular atrophy.
Pedrotti S et al.
The Embo Journal, 2010
Alternative splicing of the cyclin D1 proto-oncogene is regulated by the RNA-binding protein Sam68.
Paronetto MP et al.
Cancer Research, 2010
The germ cell nuclear proteins hnRNP G-T and RBMY activate a testis-specific exon.
Liu Y et al.
Plos Genetics, 2009
The RNA-binding and adaptor protein Sam68 modulates signal-dependent splicing and transcriptional activity of the androgen receptor.
Rajan P et al.
The Journal of Pathology, 2008
The RNA-binding protein Sam68 modulates the alternative splicing of Bcl-x.
Paronetto MP et al.
The Journal of Cell Biology, 2007
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