Associated Papers
[Inborn errors of high-density lipoprotein metabolism].
von Eckardstein A
Der Internist, 2019
Role of apolipoproteins, ABCA1 and LCAT in the biogenesis of normal and aberrant high density lipoproteins.
Zannis VI et al.
Journal of Biomedical Research, 2017
Natural human apoA-I mutations L141RPisa and L159RFIN alter HDL structure and functionality and promote atherosclerosis development in mice.
Tiniakou I et al.
Atherosclerosis, 2015
The Effect of Natural LCAT Mutations on the Biogenesis of HDL.
Fotakis P et al.
Biochemistry, 2015
Characterization of high density lipoprotein particles in familial apolipoprotein A-I deficiency.
Santos RD et al.
Journal of Lipid Research, 2008
Discrete roles of apoA-I and apoE in the biogenesis of HDL species: lessons learned from gene transfer studies in different mouse models.
Zannis VI et al.
Annals of Medicine, 2008
LCAT can rescue the abnormal phenotype produced by the natural ApoA-I mutations (Leu141Arg)Pisa and (Leu159Arg)FIN.
Koukos G et al.
Biochemistry, 2007
Cross-linking and lipid efflux properties of apoA-I mutants suggest direct association between apoA-I helices and ABCA1.
Chroni A et al.
Biochemistry, 2004
Structural and functional properties of human plasma high density-sized lipoprotein containing only apoE particles.
Krimbou L et al.
Journal of Lipid Research, 2003
Lipid-free structure and stability of apolipoprotein A-I: probing the central region by mutation.
Gorshkova IN et al.
Biochemistry, 2002
Recombinant proapoA-I(Lys107del) shows impaired lipid binding associated with reduced binding to plasma high density lipoprotein.
Huang W et al.
Atherosclerosis, 2001
Genetic determinants of high density lipoprotein levels.
Funke H
Current Opinion in Lipidology, 1997
A natural apolipoprotein A-I variant, apoA-I (L141R)Pisa, interferes with the formation of alpha-high density lipoproteins (HDL) but not with the formation of pre beta 1-HDL and influences efflux of cholesterol into plasma.
Miccoli R et al.
Journal of Lipid Research, 1997
High density lipoproteins, reverse transport of cholesterol, and coronary artery disease. Insights from mutations.
Assmann G et al.
Circulation, 1993
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