Associated Papers
Hypertrophic cardiomyopathy mutations increase myofilament Ca2+ buffering, alter intracellular Ca2+ handling, and stimulate Ca2+-dependent signaling.
Robinson P et al.
The Journal of Biological Chemistry, 2018
29760186 (PubMed )
Supplement: supp_RA118.002081_135532_2_supp_133838_p8gxk6.pdf
Two clusters of residues at the docking groove of mitogen-activated protein kinases differentially mediate their functional interaction with the tyrosine phosphatases PTP-SL and STEP.
Tárrega C et al.
The Journal of Biological Chemistry, 2002
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