Associated Papers
Photodynamic studies reveal rapid formation and appreciable turnover of tau inclusions.
Croft CL et al.
Acta Neuropathologica, 2021
Ectopic Expression Induces Abnormal Somatodendritic Distribution of Tau in the Mouse Brain.
Kubo A et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience, 2019
TIA1 regulates the generation and response to toxic tau oligomers.
Jiang L et al.
Acta Neuropathologica, 2019
APP/Go protein Gβγ-complex signaling mediates Aβ degeneration and cognitive impairment in Alzheimer's disease models.
Bignante EA et al.
Neurobiology of Aging, 2018
Depletion of Progranulin Reduces GluN2B-Containing NMDA Receptor Density, Tau Phosphorylation, and Dendritic Arborization in Mouse Primary Cortical Neurons.
Longhena F et al.
The Journal of Pharmacology and Experimental Therapeutics, 2017
Human P301L-mutant tau expression in mouse entorhinal-hippocampal network causes tau aggregation and presynaptic pathology but no cognitive deficits.
Harris JA et al.
Plos One, 2012
Hyperphosphorylation of tau and neurofilaments and activation of CDK5 and ERK1/2 in PTEN-deficient cerebella.
Nayeem N et al.
Molecular and Cellular Neurosciences, 2007
Cyclin-dependent kinase inhibitors attenuate protein hyperphosphorylation, cytoskeletal lesion formation, and motor defects in Niemann-Pick Type C mice.
Zhang M et al.
The American Journal of Pathology, 2004
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