Associated Papers
A common cause of non-obstructive azoospermia: biallelic MEI1 variants and implications for infertility diagnostics.
Tan C et al.
Journal of Assisted Reproduction and Genetics, 2026
Novel biallelic MEI1 variants cause female infertility characterized by multiple pronuclei formation and aberrant embryonic development.
Jiao X et al.
Journal of Ovarian Research, 2025
41444636 (PubMed )
Supplement: 13048_2025_1890_MOESM2_ESM.xlsx › Table_S2
Summarizing the human genes and their variants causative of non-obstructive azoospermia uncovered using whole genome/exome sequencing.
Ibrahim R et al.
Reproductive Biology, 2025
Identification of the novel homozygous whole exon deletion in MEI1 underlying azoospermia and embryonic arrest in one consanguineous family.
Liu B et al.
Reproductive Sciences (thousand Oaks, Calif.), 2025
Advances in the genetic etiology of female infertility.
Gu R et al.
Journal of Assisted Reproduction and Genetics, 2024
Bi-allelic MEI1 variants cause meiosis arrest and non-obstructive azoospermia.
Zhang Y et al.
Journal of Human Genetics, 2023
Genetic variants underlying spermatogenic arrests in men with non-obstructive azoospermia.
Ozturk S
Cell Cycle (georgetown, Tex.), 2023
Novel MEI1 compound heterozygous mutation in a sporadic family associated with spermatogenetic failure.
Chen J et al.
Asian Journal of Andrology, 2023
Meiotic recombination: insights into its mechanisms and its role in human reproduction with a special focus on non-obstructive azoospermia.
Xie C et al.
Human Reproduction Update, 2022
Whole-genome sequencing identifies new candidate genes for nonobstructive azoospermia.
Malcher A et al.
Andrology, 2022
Novel biallelic mutations in MEI1: expanding the phenotypic spectrum to human embryonic arrest and recurrent implantation failure.
Dong J et al.
Human Reproduction (oxford, England), 2021
A MEI1 homozygous missense mutation associated with meiotic arrest in a consanguineous family.
Ben Khelifa M et al.
Human Reproduction (oxford, England), 2018
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