Associated Papers
Multi-omics analyses of MEN1 missense mutations identify disruption of menin-MLL and menin-JunD interactions as critical requirements for molecular pathogenicity.
Dreijerink KMA et al.
Epigenetics & Chromatin, 2022
35941657 (PubMed )
Supplement: 13072_2022_461_MOESM5_ESM.xlsx › Database_van_mutaties
Molecular and genetic mechanisms of tumorigenesis in multiple endocrine neoplasia type-1.
Guo SS et al.
Molecular Endocrinology (baltimore, Md.), 2001
Loss of heterozygosity in sporadic parathyroid tumours: involvement of chromosome 1 and the MEN1 gene locus in 11q13.
Dwight T et al.
Clinical Endocrinology, 2000
Multiple endocrine neoplasia type 1.
Pannett AA et al.
Endocrine-related Cancer, 1999
Alternative genetic pathways in parathyroid tumorigenesis.
Farnebo F et al.
The Journal of Clinical Endocrinology and Metabolism, 1999
Alterations of the MEN1 gene in sporadic parathyroid tumors.
Farnebo F et al.
The Journal of Clinical Endocrinology and Metabolism, 1998
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