Associated Papers
Modulation of human thrombopoietin receptor conformations uncouples JAK2 V617F-driven activation from cytokine-induced stimulation.
Papadopoulos N et al.
Blood, 2023
Diagnosis- and Prognosis-Related Gene Alterations in BCR::ABL1-Negative Myeloproliferative Neoplasms.
Morishita S et al.
International Journal of Molecular Sciences, 2023
37629188 (PubMed )
Supplement: ijms-24-13008-s001.zip › ijms-2555672-supplementary.pdf
Distinct genomic landscape of Chinese pediatric acute myeloid leukemia impacts clinical risk classification.
Liu T et al.
Nature Communications, 2022
35347147 (PubMed )
Supplement: 41467_2022_29336_MOESM8_ESM.xlsx › Sheet1
Triple-Negative Essential Thrombocythemia: Clinical-Pathological and Molecular Features. A Single-Center Cohort Study.
Cattaneo D et al.
Frontiers in Oncology, 2021
Novel drivers and modifiers of MPL-dependent oncogenic transformation identified by deep mutational scanning.
Bridgford JL et al.
Blood, 2020
Molecular Pathogenesis and Clinical Significance of Driver Mutations in Primary Myelofibrosis: A Review.
Alshemmari SH et al.
Medical Principles and Practice : International Journal of the Kuwait University, Health Science Centre, 2016
Molecular diagnostics of myeloproliferative neoplasms.
Langabeer SE et al.
European Journal of Haematology, 2015
Discrepancies between bone marrow histopathology and clinical phenotype in BCR-ABL1-negative myeloproliferative neoplasms associated with splanchnic vein thrombosis.
Gianelli U et al.
Leukemia Research, 2015
Detection of MPL mutations by a novel allele-specific PCR-based strategy.
Furtado LV et al.
The Journal of Molecular Diagnostics : Jmd, 2013
Clinical utility of routine MPL exon 10 analysis in the diagnosis of essential thrombocythaemia and primary myelofibrosis.
Boyd EM et al.
British Journal of Haematology, 2010
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